Inflammatory Disorders - Part B, Volume 120 explores inflammation in the immune system and how the body’s own cells or tissues may cause abnormal inflammation, thus resulting in chronic pain, redness, swelling, stiffness, and damage to normal tissues. Chapters in this updated volume include Proteins in monogenic autoinflammatory disorders, Virulence factors and their roles in periodontal disease pathogenesis, Role of Heat shock proteins in Inflammatory disorder Rheumatoid Arthritis, Novel targeted therapies for Crohn's disease: Recent progress and future perspectives, Dietary plant flavonoids in prevention of obesity and diabetes, Small RNAs in allergic diseases, Inflammation and inflammasome during ageing and age-related diseases, and more.
1. Cover image - p. ii 2. Title page - p. ii 3. Table of Contents - p. iii 4. Advances in Protein Chemistry and Structural Biology - p. i 5. Copyright - p. iv 6. Contributors - p. ix 7. Chapter One. The expanding pathways of autoinflammation: a lesson from the first 100 genes related to autoinflammatory manifestations - p. 1 8. Chapter Two. Roles of Porphyromonas gingivalis and its virulence factors in periodontitis - p. 45 9. Chapter Three. Atherosclerosis: orchestrating cells and biomolecules involved in its activation and inhibition - p. 85 10. Chapter Four. Sphingolipids as mediators of inflammation and novel therapeutic target in inflammatory bowel disease - p. 123 11. Chapter Five. Dietary plant flavonoids in prevention of obesity and diabetes - p. 159 12. Chapter Six. The impact of microRNAs on alterations of gene regulatory networks in allergic diseases - p. 237 13. Chapter Seven. Disease modifying drugs for rheumatological diseases: a brief history of everything - p. 313 14. Chapter Eight. Comprehensive in silico screening and molecular dynamics studies of missense mutations in Sjogren-Larsson syndrome associated with the ALDH3A2 gene - p. 349 15. Chapter Nine. Computational model to analyze and characterize the functional mutations of NOD2 protein causing inflammatory disorder – Blau syndrome - p. 379 16. Chapter Ten. Structural sequence evolution and computational modeling approaches of the complement system in leishmaniasis - p. 409
Inflammatory Disorders - Part B, Volume 120 explores inflammation in the immune system and how the body’s own cells or tissues may cause abnormal inflammation, thus resulting in chronic pain, redness, swelling, stiffness, and damage to normal tissues. Chapters in this updated volume include Proteins in monogenic autoinflammatory disorders, Virulence factors and their roles in periodontal disease pathogenesis, Role of Heat shock proteins in Inflammatory disorder Rheumatoid Arthritis, Novel targeted therapies for Crohn's disease: Recent progress and future perspectives, Dietary plant flavonoids in prevention of obesity and diabetes, Small RNAs in allergic diseases, Inflammation and inflammasome during ageing and age-related diseases, and more.
1. Cover image - p. ii 2. Title page - p. ii 3. Table of Contents - p. iii 4. Advances in Protein Chemistry and Structural Biology - p. i 5. Copyright - p. iv 6. Contributors - p. ix 7. Chapter One. The expanding pathways of autoinflammation: a lesson from the first 100 genes related to autoinflammatory manifestations - p. 1 8. Chapter Two. Roles of Porphyromonas gingivalis and its virulence factors in periodontitis - p. 45 9. Chapter Three. Atherosclerosis: orchestrating cells and biomolecules involved in its activation and inhibition - p. 85 10. Chapter Four. Sphingolipids as mediators of inflammation and novel therapeutic target in inflammatory bowel disease - p. 123 11. Chapter Five. Dietary plant flavonoids in prevention of obesity and diabetes - p. 159 12. Chapter Six. The impact of microRNAs on alterations of gene regulatory networks in allergic diseases - p. 237 13. Chapter Seven. Disease modifying drugs for rheumatological diseases: a brief history of everything - p. 313 14. Chapter Eight. Comprehensive in silico screening and molecular dynamics studies of missense mutations in Sjogren-Larsson syndrome associated with the ALDH3A2 gene - p. 349 15. Chapter Nine. Computational model to analyze and characterize the functional mutations of NOD2 protein causing inflammatory disorder – Blau syndrome - p. 379 16. Chapter Ten. Structural sequence evolution and computational modeling approaches of the complement system in leishmaniasis - p. 409
